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Ligand-based Drug Design: Homework

Replication of retroviruses relies on enzyme reverse transcriptase that synthesizes a double-stranded DNA intermediate using single-stranded viral RNA as a template. The DNA intermediate is integrated by the viral integrase into the genome of the host cell. The transcription of the integrated viral genome yields viral RNA molecules and proteins that are needed for the assembly of new virions. Reverse transcriptase is unique to retroviruses and thus constitutes a good target for antiviral drugs. The first AIDS drug to reach the market, Zidovudine (AZT), inhibits the reverse transcription.

While offering hope to AIDS patient when it was approved in 1987, AZT had serous drawbacks. The chemical and metabolic insability of AZT required that large dozes of drug be taken every four hours, even at nighttime. While AZT did not bind well to nuclear DNA polymerases, it had a noticable affinity against mitochondrial DNA polymerase gamma. The drug thus interfered with mitochondrial DNA synthesis and repair, especially in rapidly dividing cells, or in cells that were subject to oxidative damage. AIDS patients who took AZT often developed mitochondrial myopathy and lactic acidosis due to the accumulation of mutations in mitochondrial DNA.

A search for better HIV reverse transcriptase inhibitors quickly ensured. As documented in the paper "Correlation between preferred sugar ring conformation and activity of nucleoside analogues against human immunodeficiency virus" by Van Roey, Salerno, Chu, and Schinazi, the molecules with similar chemical structures showed wide range of antiviral activities. In particular, it appeared that the three-dimensional conformation of the analogs had a significant effect on the ability of drugs to inhibit the reverse transcription of viral RNA. Use their paper as a starting point to understand the conformational requirements for effective HIV reverse transcriptase inhibitors, and propose new structures that would show desired conformational properties while being stable toward acid-catalyzed depurination.

With the help of literature searches and/or computer modeling, answer the following questions:

Please follow this link to see homework for Winter 2011 course.


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Tutorial by Dr. Kalju Kahn, Department of Chemistry and Biochemistry, UC Santa Barbara. ©2008